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论文共 9 篇作者统计合作学者相似作者

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Editorial: Therapeutics in Pulmonary Arterial Hypertension
Rui Adão,Francisco Perez-Vizcaino,Bassam Redwan,Carmen Brás-Silva
Frontiers in cardiovascular medicine (2024): 1463305-1463305
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Urocortin-2 in Acute Heart Failure: Role As a Marker of Volume Overload and Pulmonary Hypertension
Mariana Pintalhão, C. Maia-Rocha,Paulo Castro-Chaves,Rui Adão, António S. Barros, Rosário Martíns, Adelino Leite‐Moreira, Paulo Bettencourt,Carmen Brás‐Silva
Current problems in cardiologyno. 4 (2022): 100860-100860
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Urocortina-2 Atenua a Disfunção Ventricular Direita E a Hipertensão Arterial Pulmonar
Rui Adão,Pedro Mendes‐Ferreira, C. Maia-Rocha,Diana Santos‐Ribeiro, Carlos Eduardo Pinto de Pinto, Luisa Margarita Cabrera Pimentel,François Potus, Sandra Breuils‐Bonnet, S. Provencher, Sandra Breuils Bonnet, A. Leite-Moreira,Carmen Brás‐Silva
openalex（2016）
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Mecanismos Moleculares Subjacentes Aos Efeitos Benéficos Da Neuregulina-1 No Tratamento Da Hipertensão Arterial Pulmonar
C. Maia-Rocha,Pedro Mendes‐Ferreira,Rui Adão, Débora da Silva Freitas Ribeiro, B.S. Alves, Gilles W. De Keulenaer, A. Leite-Moreira,Carmen Brás‐Silva
openalex（2014）
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Right Ventricle Pressure Overload-Induced Hypertrophy is Attenuated by Neuregulin-1
B.S. Alves,Diana Santos‐Ribeiro,Rui Adão, C. Maia-Rocha,Pedro Mendes‐Ferreira, Gilles W. De Keulenaer, A. Leite-Moreira,Carmen Brás‐Silva
openalex（2014）
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NRG-1 Improves Monocrotaline-Induced Right Ventricular and Pulmonary Endothelial Dysfunction
Pedro Mendes‐Ferreira, C. Maia-Rocha,Rui Adão, Rui Cerqueira, Maria José Mendes, A.P. Lourenco,Sónia Pinho, Gilles W. De Keulenaer, A. Leite-Moreira,Carmen Brás‐Silva
openalex（2012）
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Urocortin-2 Attenuates Pulmonary Arterial Hypertension
Rui Adão,Pedro Mendes‐Ferreira, C. Maia-Rocha,Diana Santos‐Ribeiro, Carlos Eduardo Pinto de Pinto, Luís Pimentel,François Potus, Sandra Breuils‐Bonnet, MT Rademaker, S. Provencher, Sandra Breuils Bonnet, A. Leite-Moreira,Carmen Brás‐Silva
semanticscholar
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作者统计

#Papers: 8

#Citation: 23

H-Index: 1

G-Index: 4

Sociability: 3

Diversity: 0

Activity: 0

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