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Receptor For Advanced Glycation End Products And Age-Related Macular Degeneration

INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE, no. 10 (2004): 3713-3720

Cited: 110|Views29

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academic.microsoft.com pubmed.ncbi.nlm.nih.gov

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Abstract

PURPOSE. Advanced glycation end products ( AGE) exacerbate disease progression through two general mechanisms: modifying molecules and forming nondegradable aggregates, thus impairing normal cellular/tissue functions, and altering cellular function directly through receptor-mediated activation. In the present study receptor for AGE ( RAGE...More

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