Pressure-Overload Induced Left Ventricular Hypertrophy and Dysfunction in Mice are Exacerbated by Congenital Nitric Oxide Synthase 3 Deficiency

Fumito Ichinose,Kenneth D. Bloch,Justina C. Wu,Ryuji Hataishi, H. Thomas

msra（2003）

引用 23|浏览3

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摘要

Abstract To investigate the role of endothelial nitric oxide synthase (NOS3) in left ventricular (LV) remodeling induced by chronic pressure-overload, the impact of transverse aortic constriction (TAC) on LV structure and function was compared,in wild-type (WT) and NOS3-deficient (NOS3

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