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Keratinocyte Differentiation Marker Suppression by Arsenic: Mediation by AP1 Response Elements and Antagonism by Tetradecanoylphorbol Acetate

Toxicology and Applied Pharmacology, no. 3 (2001): 302-311

Cited by: 32|Views3
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Abstract

Culture models of target cells are anticipated to help elucidate the mechanism by which inorganic arsenic acts as a carcinogen in humans. Present work characterizes the response of human keratinocytes, a target cell type, to arsenic suppression of their differentiation program. Four representative differentiation marker mRNAs (involucrin,...More

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