Overexpression of filamin c in chronic intermittent hypoxia-induced cardiomyocyte apoptosis is a potential cardioprotective target for obstructive sleep apnea.

Xuechao Yang,Yang Shi,Linfei Zhang,Huan Liu,Yongfeng Shao,Shijiang Zhang

Sleep & breathing = Schlaf & Atmung（2019）

引用 7|浏览20

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摘要

These results suggested FLNC is implicated in the pathogenesis of CIH-induced cardiomyocyte apoptosis, and FLNC may serve as a novel cardioprotective target for OSA patients.

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关键词

Cardiomyocyte apoptosis,Chronic intermittent hypoxia,FLNC,Obstructive sleep apnea

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