Dexmedetomidine postconditioning attenuates myocardial ischemia/reperfusion injury by activating the Nrf2/Sirt3/SOD2 signaling pathway in the rats.

Bin Hu,Tian Tian,Xin-Tao Li,Pei-Pei Hao,Wei-Chao Liu,Ying-Gui Chen,Tian-Yu Jiang,Pei-Shan Chen,Yi Cheng,Fu-Shan Xue

Redox report : communications in free radical research（2023）

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摘要

Dex postconditioning can alleviate myocardial IRI by suppressing oxidative stress and apoptosis, and these beneficial effects are at least partly mediated by activating the Nrf2/Sirt3/SOD2 signaling pathway.

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关键词

Dexmedetomidine,Nrf2/Sirt3/SOD2 signaling pathway,apoptosis,myocardial ischemia/reperfusion injury,oxidative stress

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