Cross-talk between the RAS-ERK and mTOR signalings-associated autophagy contributes to tripterygium glycosides tablet-induced liver injury.
Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie(2023)
摘要
RAS-ERK-mTOR cross-talk may play a crucial role in TGT-induced hepatocyte autophagy, offering a promising target for developing novel therapeutics to combat TGT-induced hepatotoxicity.
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关键词
Autophagy,Drug-induced liver injury,RAS-ERK-mTOR cross-talk,Tripterygium glycosides tablet,UPLC-Q/TOF-MS/MS
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