Not just snot: local inflammatory profiles in patients with aspirin-exacerbated respiratory disease differ from patients with aspirin-tolerant chronic rhinosinusitis

Simon Chiang, Sophie E. E. Yu, Tejas S. Athni,Regan W. Bergmark,Alice Z. Maxfield,Rachel E. Roditi,Kathleen M. Buchheit,Marie Lundberg,Margaret B. Mitchell,Stella E. E. Lee

INTERNATIONAL FORUM OF ALLERGY & RHINOLOGY（2024）

引用 0|浏览6

暂无评分

摘要

IL-5, CCL2, and CXCL8 in sinus mucous are higher in patients with AERD relative to aspirin-tolerant patients with CRS These mediators are pleiotropic, leading to widescale inflammatory processes contributing to AERD AERD is not only a T2 disease but heterogeneous: this may explain the refractory nature of AERD.

查看译文

关键词

asthma,biomarker,chronic rhinosinusitis

AI 理解论文

溯源树

样例

生成溯源树，研究论文发展脉络

Chat Paper

正在生成论文摘要