
Evidence on virtual surgical planning (VSP), patient-specific implants (PSI), and three-dimensional (3D) printing in mandibular reconstruction comes largely from oncologic or mixed-etiology cohorts, although patients with ameloblastoma are younger, are not irradiated, and need long-term surveillance and dental rehabilitation. This review combined a bibliometric analysis of Scopus records (2010–2026) with an etiology-stratified narrative review. Records were coded into eight etiology categories by a language model checked against manual coding (κ = 0.78), and 78 clinical studies were classified as ameloblastoma-specific (n = 16), mixed etiology with extractable ameloblastoma data (n = 35), or mixed etiology without separate data (n = 27). Annual output rose from 49 documents in 2010 to 272 in 2025. Of 2404 digital-technology records, 5.4% were ameloblastoma-specific and 10.2% oncologic only. Ameloblastoma-specific records had a lower mean normalized citation score than oncologic records (0.52 vs 1.05; P < .001) and less often cited the core VSP accuracy literature (29.7% vs 68.5%; P < .001). Of the 78 clinical studies, 67 were at Oxford level 4 or 5; none of the 11 level 2–3 studies, all mixed-etiology cohorts, analyzed ameloblastoma separately, and only one study averaged >5 years of follow-up. Mixed cohorts suggest better condylar positioning and possibly shorter operative time, benefits that depend mainly on defect and technique, but recurrence, rehabilitation, quality of life, and cost were rarely measured. The evidence supports selective rather than routine use of digital workflows in ameloblastoma. Diagnosis-specific reporting, follow-up beyond 5 years, and a core outcome set are needed.
Introduction To correlate the immediate skeletal and occlusal pattern changes with the electromyographic (EMG) activity of anterior temporalis and superficial masseter muscles following rapid maxillary expansion (RME). Material and Methods The study included 12 patients aged 10–15 years (mean, 12.5 ± 1.45 years) who underwent RME therapy using a standardised protocol. EMG of superficial masseter and anterior temporalis and T-scans were taken before appliance fixation (T0), immediately after the appliance fixation (T1), three weeks of expansion (T2), and three months post-expansion (T3). Changes in the occlusal pattern and muscle activity following rapid palatal expansion were measured using the Friedman analysis of variance (ANOVA) test and correlated using Pearson's correlation coefficient. Result The occlusal pattern changes following RME are significant at molars, i.e., the P-Value for right side molars is significant, i.e., P = 0.042, and non-significant at premolars. The changes in EMG activity of superficial masseter and anterior temporalis over three months following RME, i.e.(P > 0.05), were non-significant. Conclusion The muscular activity of the masseter and temporalis muscles following RME increases transiently, then decreases toward baseline due to neuromuscular adaptation. The muscular activity changes of the masseter and temporalis are weak and negatively correlated to the occlusal pattern changes following rapid maxillary expansion.
Osseous choristoma is a rare benign lesion composed of mature bone in an ectopic soft-tissue site, most often the posterior tongue. We report a 9-year-old girl with a 1-year history of a slowly enlarging tongue-base lesion causing dysphagia and snoring during sleep. Contrast-enhanced computed tomography showed a well-circumscribed, homogeneous bony-density lesion measuring 4.0 & times; 3.8 & times; 4.1 cm without enhancement or adjacent soft-tissue invasion. The lesion was completely excised through a dorsal tongue approach. Histopathological examination demonstrated mature osseous tissue with bony trabeculae and marrow cavities, confirming osseous choristoma. Recovery was uneventful, with no clinical recurrence at 3 months. Large tongue-base osseous choristomas may cause functional symptoms despite their benign nature. Computed tomography provides a useful diagnostic clue, while definitive diagnosis depends on histopathology. Complete surgical excision remains the treatment of choice.