Transient decrease of serum iron after acute erythropoietin treatment contributes to hepcidin inhibition by ERFE in mice.

Irene Artuso,Mariateresa Pettinato,Antonella Nai,Alessia Pagani,Ugo Sardo,Benjamin Billoré,Maria Rosa Lidonnici,Cavan Bennett,Giacomo Mandelli,Sant-Rayn Pasricha,Giuliana Ferrari,Clara Camaschella,Léon Kautz,Laura Silvestri

HAEMATOLOGICA（2019）

引用 22|浏览23

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摘要

To coordinate iron supply with hemoglobin (Hb) synthesis, erythropoietin (EPO)-stimulated erythroid precursors release erythroferrone (ERFE), a soluble protein that suppresses the expression of hepcidin ( Hamp ).[1][1] Hepcidin is a short liver peptide that controls iron entry into the circulation

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关键词

BMP-SMAD pathway,H3K4me3,Iron Metabolism,erythroferrone,hepcidin

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