Interleukin-33 Exacerbates IgA Glomerulonephritis in Transgenic Mice Overexpressing B Cell Activating Factor.

Yuan Min Wang, Karli Shaw,Geoff Yu Zhang,Edmund Y M Chung,Min Hu,Qi Cao,Yiping Wang,Guoping Zheng,Huiling Wu,Steven J Chadban,Hugh J McCarthy,David C H Harris,Fabienne Mackay,Shane T Grey,Stephen I Alexander

Journal of the American Society of Nephrology : JASN（2022）

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摘要

IL-33-expanded ILC2s exacerbated IgA glomerulonephritis in a mouse model. These findings indicate that IL-33 and ILC2s warrant evaluation as possible mediators of human IgA nephropathy.

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关键词

B cell activating factor,IL-33,IgA glomerulonephritis,innate lymphoid cells 2,mice,suppression of tumorigenicity 2 (ST2),transgenic

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