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p16 INK4A drives nonalcoholic fatty liver disease phenotypes in high fat diet fed mice through biliary E2F1/FOXO1/IGF-1 signaling.

Debjyoti Kundu,Lindsey Kennedy,Tianhao Zhou,Burcin Ekser,Vik Meadows,Amelia Sybenga,Konstantina Kyritsi,Lixian Chen,Ludovica Ceci,Nan Wu,Chaodong Wu,Shannon Glaser,Guido Carpino,Paolo Onori,Eugenio Gaudio,Gianfranco Alpini,Heather Francis

Hepatology (Baltimore, Md.)（2023）

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摘要
Attenuating hepatic p16 expression may be a therapeutic approach for improving NAFLD/NASH phenotypes.
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关键词
biliary e2f1/foxo1/igf-1,liver
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