Repurposing FBP1: dephosphorylating IB to suppress NFB
CELL RESEARCH(2023)
摘要
Metabolic enzymes can function as protein kinases and protein phosphatases to govern the phosphorylation status of protein substrates, thereby regulating pivotal cellular activities. A new study published in Cell Research demonstrated that fructose 1,6-bisphosphatase 1 directly dephosphorylates I kappa B alpha to suppress NF kappa B activation and modulate the tumor immune microenvironment.
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