FABP4 in LSECs promotes CXCL10-mediated macrophage recruitment and M1 polarization during NAFLD progression.

Cui Zhou,Zhenyang Shen,Bo Shen,Weiming Dai, Zhongsang Sun,Yuecheng Guo,Xianjun Xu,Junjun Wang,Jingyi Lu,Qingqing Zhang,Xin Luo,Ying Qu,Hui Dong,Lungen Lu

Biochimica et biophysica acta. Molecular basis of disease（2023）

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摘要

FABP4 in LSECs may play a pathogenic role in NAFLD course by promoting CXCL10-mediated macrophage M1 polarization and CXCR3 macrophage infiltration via activating NF-κB/p65 signaling.

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关键词

ALT,AST,ELISA,FABP4,FDG,GGT,HDL-c,H&E,HFD,IF,IHC,KCs,LSECs,LSM,MNI,LDL-c,NAFL,NAFLD,NASH,NFD,NPCs,ORO,PA,qRT-PCR,TC,TG,UAP

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