TCFL5 knockdown sensitizes DLBCL to doxorubicin treatment via regulation of GPX4.
Cellular signalling（2023）
摘要
DLBCL cells overexpress TCFL5 that promotes chemoresistance by regulating GPX4. Targeting TCFL5 may provide a prospective therapeutic strategy for doxorubicin-resistant DLBCL.
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关键词
Drug resistance, Doxorubicin, DLBCL, TCFL5, GPX4
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