Methyltransferase-like 3 facilitates the stem cell properties of esophageal cancer by upregulating patched homolog 1 via N6-methyladenosine methylation
American Journal of Physiology-cell Physiology（2023）
摘要
PTCH1 has been proved to facilitate cell proliferation and self-renewal in esophageal cancer. We studied the upstream regulation mechanism of PTCH1 by METTL3 through m 6 A modification. Our results provide a new target and theoretical basis for the treatment of esophageal cancer.
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关键词
esophageal cancer,methylation,stem cell properties,methyltransferase-like
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