Case Reports1 January 1937PRIMARY PNEUMOCOCCIC PERITONITIS; RECOVERY OF THE ACUTE SEROUS TYPE FOLLOWING TYPE I SERUM TREATMENT WITHOUT SURGICAL INTERVENTIONMANUEL M. GLAZIER, M.D., BERNARD I. GOLDBERG, M.D., A. A. WEINSTEIN, M.D.MANUEL M. GLAZIER, M.D.Search for more papers by this author, BERNARD I. GOLDBERG, M.D.Search for more papers by this author, A. A. WEINSTEIN, M.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-10-7-1042 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptPneumococcic peritonitis is comparatively uncommon and bears a grave prognosis. It is generally divided into two types, primary and secondary. The primary type is characterized by involvement of the peritoneum without any discernible focus of infection and it tends to run one of two courses. The most common course is an acute diffuse serous peritonitis that usually ends quickly in exitus. The second course results in a localized and circumscribed abscess of the peritoneum and is associated with a more favorable prognosis. In the secondary type of pneumococcic peritonitis we find an antecedent or coincidental source of infection, usually the...Bibliography1. LIPSHUTZLOWENBURG BH: Pneumococcic and streptococcic peritonitis, Jr. Am. Med. Assoc., 1926, lxxxvi, 99-104. CrossrefGoogle Scholar2. JENSEN J: Pneumococcus Peritonitis, Arch. f. klin. Chir., 1903, lxix, 1134-1157. Google Scholar3. WOLFSOHN G: Über pneumokokken Peritonitis, Med. Klin., 1925, xxi (2), 1638-1642. Google Scholar4. MCCARTNEYFRASER JEJ: Pneumococcal peritonitis, Brit. Jr. Surg., 1921-1922, ix, 479-489. Google Scholar5. OBADALEK W: Die Frühoperation der pneumokokken Peritonitis im Kindesalter, Zentralbl. f. Chir., 1931, lviii, 1250-1258. Google Scholar6. KOENNECKE W: Über Pneumokokkenperitonitis, Beitr. z. klin. Chir., 1919, cxv, 408-428. Google Scholar7. RISCHBIETH H: On pneumococcus peritonitis, Quart. Jr. Med., 1910-1911, iv, 205-231. Google Scholar8. ROLLESTON HD: Pneumococcic peritonitis, Clin. Jr., 1908, xxxi, 319-320. Google Scholar9. ELKIN DC: Pneumococcus peritonitis, Arch. Surg., 1929, xviii, 745-752. CrossrefGoogle Scholar10. BLAKECECIL FGRL: Treatment of experimental pneumococcus Type I pneumonia in monkeys with Type I antipneumococcus serum, Jr. Exper. Med., 1920, xxxii, 1-18. Google Scholar11. PEISER A: Über das Verhalten der serösen Körperhöhlen gegenüber im Blute Kreisenden Bakterien, Beitr. z. klin. Chir., 1907, lv, 484-495. Google Scholar12. BARRINGTON-WARD LE: Pneumococcal and streptococcal peritonitis, Brit. Med. Jr., 1932, ii, 257-258. Google Scholar13. KRAMER E: Über die abortive Form der pneumokokken Peritonitis, Monatsschr. f. Kinderheilk., 1935, lxi, 370-373. Google Scholar14. NEUHOFFCOHEN HJ: Abdominal puncture in the diagnosis of acute intraperitoneal disease, Ann. Surg., 1926, lxxxiii, 454-462. CrossrefGoogle Scholar15. LOEWE O: Die Schnelldiagnose der pneumokokken Peritonitis, Zentralbl. f. Chir., 1932, lix, 3049-3051. Google Scholar16. LEONARDO RA: Primary pneumococcus peritonitis, Ann. Surg., 1926, lxxxiii, 411-416. CrossrefGoogle Scholar17. SCHAUDIG H: Zur Behandlung der Pneumokokkenperitonitis, Zentralbl. f. Chir., 1933, lx, 1344-1346. Google Scholar18. STRUTHER RR: Pneumococcal peritonitis, Canad. Med. Assoc. Jr., 1924, xiv, 955-959. Google Scholar19. FRICKE E: Pneumococcus peritonitis, Am. Jr. Surg., 1930, viii, 48-53. CrossrefGoogle Scholar20. GLASS E: Zur konservativen Behandlung der Pneumokokkenperitonitis, Zentralbl. f. Chir., 1933, lx, 2430-2432. Google Scholar21. GIBSON CL: Pneumococcus peritonitis, Surg. Clin. N. Am., 1925, v, 183-186. Google Scholar22. LAZARUS SD: Acute peritonitis in childhood, Am. Jr. Surg., 1932, lxvii, 70-73. CrossrefGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAuthors: MANUEL M. GLAZIER, M.D.; BERNARD I. GOLDBERG, M.D.; A. A. WEINSTEIN, M.D.Affiliations: Boston, Massachusetts*Received for publication June 17, 1936.From the Departments of Pediatrics and Medicine, Beth Israel Hospital and Tufts College Medical School. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byPneumococcus peritonitis in nephrotic and non-nephrotic children 1 January 1937Volume 10, Issue 7Page: 1042-1049KeywordsAbscessesHospital medicinePediatricsPeritoneumPeritonitis ePublished: 1 December 2008 Issue Published: 1 January 1937 PDF downloadLoading ...

S TUDIES of the heart in spontaneous myxedema”. lo, ‘l, “‘j ‘*, “7 3’ nuring the past decade have demonstrated that (1) the heart size, as measured by the seven-foot roentgenogram, is increased; (2) the voltages of the P, T, and QRS waves of the electrocardiogram are frequently diminished *18p 311 331 35, 3g and (3) cardiac contractions are less vigorous.2’ 3o Opinions differ concerning the clinical significance of these alterations. Zondek42* 43 and Fahr14 maintain that cardiac function is often impaired in patients with myxedema having such changes. Christian,lly I2 Willius and Haines,41 Case,lO Means, White and Krantz,28 however, studied a total of three hundred patients with myxedema and concluded that heart function is rarely, if ever, impaired. From a recent review of the literature and a comprehensive study of thirty additional cases at the Massachusetts General Hospital, Lerman, Clark and Means?’ conclude that “myxedema heart” in the sense of heart failure occurs rarely, if at all. The studies of the above investigators were confined almost entirely to patients without cardiovascular disease. In treating patients with chronic heart disease and other conditions by inducing hypothyroidism by total removal of the normal thyroid gland, we have been able to study the development of the cardiovascular changes associated with the development of myxedema. Two aspects of the heart in myxedema have been investigated: first, the character and rate of development of the changes in heart size and electrocardiographic tracings; and, second, the significance of these changes in terms of cardiac function. Studies before, and at varying intervals aft.er, total thyroidectomy have been ma.de in three groups of patients: one group comprising patients with congestive heart failure at the time of, or just before, operation; the second group, patients with angina pectoris ; and the third group, patients with no evident functional or anatomical abnormalities. The rationale, t,eehnie, and therapeutic results of total thyroidectomy in patients with chronic heart disease and no thyrotoxicosis have been described in previous communications.4* 8l gl 15, I63 37