Renal allotransplantation is the treatment of choice for patients with end-stage renal disease (ESRD). With increasing technical expertise, it has been realised that presence of a normal lower urinary tract is not a necessity for transplantation. Advanced genito-urinary tuberculosis frequently leads to a small-capacity bladder. Herein, we present a case of ESRD due to urinary tuberculosis in whom sigmoid colon conduit urinary diversion was carried out. The immunosuppressed status did not increase the complications of a sigmoid colon conduit urinary diversion.

International Journal of Clinical PracticeVolume 41, Issue 7 p. 839-840 Clinical Study Student electives in tropical countries — the incidence of clinical signs in an African general hospital A D Harries MD, MRCP, DTM&H, Corresponding Author A D Harries MD, MRCP, DTM&H Lecturer n/[email protected] Department of Medicine, University of Maiduguri, Maiduguri, Nigeria On secondment from Liverpool School of Tropical Medicine.Address for correspondence: Dr A D Harries, Department of Medicine, Kamuzu Central Hospital, PO Box 149, Lilongwe, Malawi.Search for more papers by this authorS K Hoggar MB, CHB, S K Hoggar MB, CHB Senior Medical Officer Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorA C Nwosu MB BS, A C Nwosu MB BS Registrar Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorK S Chugh MD, FICA, K S Chugh MD, FICA Professor Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this author A D Harries MD, MRCP, DTM&H, Corresponding Author A D Harries MD, MRCP, DTM&H Lecturer n/[email protected] Department of Medicine, University of Maiduguri, Maiduguri, Nigeria On secondment from Liverpool School of Tropical Medicine.Address for correspondence: Dr A D Harries, Department of Medicine, Kamuzu Central Hospital, PO Box 149, Lilongwe, Malawi.Search for more papers by this authorS K Hoggar MB, CHB, S K Hoggar MB, CHB Senior Medical Officer Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorA C Nwosu MB BS, A C Nwosu MB BS Registrar Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorK S Chugh MD, FICA, K S Chugh MD, FICA Professor Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this author First published: 01 July 1987 https://doi.org/10.1111/j.1742-1241.1987.tb08488.x Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL No abstract is available for this article. Volume41, Issue7July 1987Pages 839-840 RelatedInformation

International Journal of Clinical PracticeVolume 41, Issue 3 p. 655-658 Clinical Study Aphthous ulceration in Nigerian and British medical students A D Harries MD, MRCP, A D Harries MD, MRCP Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorP Mohammed, P Mohammed Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorK S Chugh MD, FICA, K S Chugh MD, FICA Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorD Burnell MB, D Burnell MB Department of Gastroenterology, University Hospital of Wales, Heath Park, CardiffSearch for more papers by this authorJ Rhodes MD, FRCP, Corresponding Author J Rhodes MD, FRCP n/[email protected] Department of Gastroenterology, University Hospital of Wales, Heath Park, CardiffCorrespondence to: Dr J Rhodes, Consultant Physician, University Hospital of Wales, Heath Park, Cardiff CF4 4XW.Search for more papers by this author A D Harries MD, MRCP, A D Harries MD, MRCP Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorP Mohammed, P Mohammed Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorK S Chugh MD, FICA, K S Chugh MD, FICA Department of Medicine, University of Maiduguri, Maiduguri, NigeriaSearch for more papers by this authorD Burnell MB, D Burnell MB Department of Gastroenterology, University Hospital of Wales, Heath Park, CardiffSearch for more papers by this authorJ Rhodes MD, FRCP, Corresponding Author J Rhodes MD, FRCP n/[email protected] Department of Gastroenterology, University Hospital of Wales, Heath Park, CardiffCorrespondence to: Dr J Rhodes, Consultant Physician, University Hospital of Wales, Heath Park, Cardiff CF4 4XW.Search for more papers by this author First published: 01 March 1987 https://doi.org/10.1111/j.1742-1241.1987.tb08081.xRead the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Volume41, Issue3March 1987Pages 655-658 RelatedInformation

In the past fifteen years malaria has returned to several areas from where it had been totally eradicated. According to recent estimates about 300 million people in the world develop malaria each year and about a million die in tropical Africa alone (1). Renal involvement in malaria had been suspected for a long time on the basis of a higher prevalence of renal disease in endemic areas (2), but it was only in 1930 that Giglioli, working in Guyana in South America, obtained sufficient evidence to postulate a cause and effect relationship between Plasmodium malariae infection and the nephrotic syndrome (3). Subsequent observation by others have confirmed this association (4-6). Glomerular lesions have been documented both in Plasmodium falciparum and P. malariae infections. In falciparum malaria, a "transient" glomerulonephritis often ocurs but mostly goes undetected because of the absence of hypertension, edema or any decline in renal function (7, 8). A nephrotic syndrome has also been described (9). Proteinuria, usually nonselective and less than 1 gm per 24 hours, microscopic hematuria, and cylindruria are seen in 2050% of patients. The urinary abnormalities disappear within 2-3 weeks of the onset of infection. During the acute phase, serum C3 and C4 are decreased, and P. falciparum-soluble antigen, antibodies to P. falciparum, and circulating immune complexes are demonstrable (7, 8, 10). Renal biopsy reveals widening of the mesangial area, mesangial and endothelial cell proliferation, and pigment laden macrophages and eosinophilic granular material in the capillary lumina (8, 11). Immunofluorescence microscopy reveals granular deposits of IgM and C3 chiefly in the mesangium. This form of glomerular injury is reversible with anti-malarial therapy and does not lead to chronic renal disease (11). In contrast, the glomerular lesion in quartan rnaThe International Journal Of Artificial Organs / Vol. 9 no. 6, 1986/ p.p. 391-392

Two hundred and fifty-one Nigerian medical and nursing students from the University of Maiduguri Teaching Hospital completed a questionnaire on chloroquine-induced pruritus. One hundred and thirty (51.8) students experienced itching, and in 71 the symptoms were so distressing that chloroquine was no longer used for treating febrile malarial episodes. Antihistamines taken before chloroquine or a change in the route of administration were helpful in preventing or a ameliorating symptoms in some of the students. Significantly more students with chloroquine-induced pruritus had parents or siblings similarly affected compared with students who did not get pruritus, and this supports the concept of a pharmacogenetic basis to the disorder.

Rupture of the renal allograft, although a rare complication, was encountered in 11 of our 152 renal transplants. The commonest cause was associated acute rejection. We treated our patients conservatively by repair of the rupture and antirejection therapy. 8 of 11 kidneys could be saved by this method. The patient survival was 8 out of 11. Deaths were unrelated to the graft rupture.

"Snake bite: frequency of adult admissions to a general hospital in north-east Nigeria." Annals of Tropical Medicine & Parasitology, 78(6), pp. 665–666

A 37-yr-old man developed acute glomerulonephritis in association with filariasis. Renal histology revealed varying degrees of mesangial cell hypercellularity.