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We study birth defects and regeneration. Organogenesis is regulated by several inductive factors, such as Wnts. Incorrect activity and timing of these signaling pathways during early development frequently result in embryonic death or severe birth defects. We are investigating the mechanism and prevention of birth defects mainly using Wnt signaling mutant mice as the research model. We are also investigating the role of Wnt signaling in stem cell/progenitor renewal and regeneration processes in order to enhance their therapeutic potential.
From the aspect of congenital disease, we are investigating the mechanism and prevention of several major birth defects related to tissue closure and patterning in Wnt signaling mutant animal models, such as: Orofacial cleft (OFC), defective tissue closure in the upper lip, palate, and related orofacial structures, including cleft lip with or without cleft palate (CLP) and isolated cleft palate; Neural tube defect (NTD), defective closure of the neural tube (the precursor of brain and spinal cord), including spina bifida (open spine) and exencephaly (open skull/brain); and other congenital disorders in several major organs, including the brain, heart, and kidney.
From the aspect of stem cell biology, we are addressing the role of Wnt signaling in tissue/organ-specific stem cells such as neural crest cells, neural stem cells or radial glia during development and regeneration.
研究兴趣
论文共 67 篇作者统计合作学者相似作者
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Frontiers in physiology (2023)
Birth defects researchno. 19 (2023): 1851-1865
Birth defects researchno. 19 (2023): 1835-1850
Chao Li,Yuming Feng, Zhenxin Fu, Junjie Deng,Yue Gu,Hanben Wang,Xin Wu,Zhengyun Huang,Yichen Zhu,Zhiwei Liu,Moli Huang,Tao Wang,Shijun Hu,Bing Yao,Yizhun Zeng,Chengji J. Zhou,Steve D. M. Brown,Yi Liu,Antonio Vidal-Puig,Yingying Dong,Ying Xu
npj regenerative medicineno. 1 (2022)
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#Papers: 65
#Citation: 2209
H-Index: 30
G-Index: 46
Sociability: 5
Diversity: 3
Activity: 20
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