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Dr. Rachel Perry is an Assistant Professor in Medicine/Endocrinology and Cellular & Molecular Physiology at the Yale University School of Medicine. Rachel's background is in the use of hyperinsulinemic-euglycemic clamps and stable isotope infusions to assess insulin sensitivity, having earned her B.S. in Biomedical Engineering, Ph.D. (with Distinction) in Cellular & Molecular Physiology, and performed her postdoctoral training in Medicine/Endocrinology, all in the laboratory of Dr. Gerald Shulman. Rachel's CV includes first-author papers in Nature, Cell (2), Science, JCI, PNAS, Nature Medicine (2), PNAS, Nature Communications (3), JBC, Cell Metabolism (2), and AJP-Endocrinology.
The Perry laboratory focuses on applying stable isotope tracer methods to understand obesity- and insulin-associated alterations in metabolic flux pathways. Dr. Perry and her colleagues have recently identified hyperinsulinemia-induced increases in tumor glucose uptake and oxidation as a critical driver of colon cancer in two mouse models of the disease, and mitochondrial uncoupling as a potential therapeutic strategy against the disease (Wang et al. Cell Reports 2018, Nasiri et al. Cancer & Metabolism 2019), and went on to show that responsiveness to insulin is a metabolic signature of obesity-associated tumor types in vitro (Rabin-Court et al. PLoS One 2019).
Dr. Rachel Perry is an Assistant Professor in Medicine/Endocrinology and Cellular & Molecular Physiology at the Yale University School of Medicine. Rachel's background is in the use of hyperinsulinemic-euglycemic clamps and stable isotope infusions to assess insulin sensitivity, having earned her B.S. in Biomedical Engineering, Ph.D. (with Distinction) in Cellular & Molecular Physiology, and performed her postdoctoral training in Medicine/Endocrinology, all in the laboratory of Dr. Gerald Shulman. Rachel's CV includes first-author papers in Nature, Cell (2), Science, JCI, PNAS, Nature Medicine (2), PNAS, Nature Communications (3), JBC, Cell Metabolism (2), and AJP-Endocrinology.
The Perry laboratory focuses on applying stable isotope tracer methods to understand obesity- and insulin-associated alterations in metabolic flux pathways. Dr. Perry and her colleagues have recently identified hyperinsulinemia-induced increases in tumor glucose uptake and oxidation as a critical driver of colon cancer in two mouse models of the disease, and mitochondrial uncoupling as a potential therapeutic strategy against the disease (Wang et al. Cell Reports 2018, Nasiri et al. Cancer & Metabolism 2019), and went on to show that responsiveness to insulin is a metabolic signature of obesity-associated tumor types in vitro (Rabin-Court et al. PLoS One 2019).
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Gautham Ramshankar,Rachel J Perry
Diabetesno. 3 (2024): 357-358
Matrix Biology (2024): 38-47
American journal of physiology. Endocrinology and metabolismno. 3 (2024): E290-E307
Ngan K. Vu,Rachel J. Perry
Clinical and Translational Discoveryno. 2 (2024): n/a-n/a
Tumininu S Faniyan,Xinyi Zhang,Donald A Morgan,Jorge Robles,Siresha Bathina, Paul S Brookes,Kamal Rahmouni,Rachel J Perry,Kavaljit H Chhabra
bioRxiv : the preprint server for biology (2024)
Nature Metabolismpp.1-23, (2024)
Tumininu S. Faniyan,Xinyi Zhang,Donald A. Morgan, Jorge Robles,Siresha Bathina, Paul S. Brookes,Kamal Rahmouni,Rachel J. Perry, Kavaljit H. Chhabra
crossref(2024)
ENDOCRINOLOGYno. 4 (2023)
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