目的:检验血液流变学和血小板功能是否与原发性高血压(EHTs)伴代谢综合征(Mets)有关.方法:原发性高血压为世界卫生组织WHO规定I级.测定133例未经治疗的原发性高血压患者中,测定血液粘度和血小板表面标记物.结果:43例伴代谢综合征患者的血液粘度与不伴代谢综合征的91例相比,显著提高.血细胞比容和血浆粘度在伴代谢综合征组中升高,尽管红细胞刚性指数“k”在各组并无不同.作为一个整体,血液粘度与红细胞压积和血浆粘度密切相关.另外,血浆粘度与甘油三酯密切相关,内稳态评估指数、C反应蛋白和血浆纤维蛋白有明确关联,与高密度脂蛋白呈负相关.各组中血小板表面标记物并无不同.结论:血浓缩和血浆粘度增加而引起的血液粘度增加,与早期原发性高血压伴代谢综合征具有相关性.

目的探讨急性心肌梗死(Acute myocardial infarction,AMI)患者溶栓治疗后血清同型半胱氨酸(Homocysteine,Hcy)水平的变化及对愈后的意义。方法应用酶连免疫法对40例急AMI患者进行了溶栓治疗前后血浆Hcy的检测,并与40例正常健康人进行比较。结果在溶栓治疗前,急性心梗患者血浆Hcy水平显著高于对照组(P<0.01);治疗后2周,则与对照组比较无显著性差异(P>0.05)。结论检测AMI患者血浆Hcy水平的变化,对了解病情、观察预后有重要的临床价值。

Objective To discuss the etiology of pulmonary embolism.Methods Retrospective analysis of 17 patients with pulmonary thromboembolism,carries on the analysis and the discussion of the pathogenesis.Results Pulmonary Thromboembolism Etiology and more complicated,in which thrombotic deep thrombophlebitis and deep vein thrombosis is the most common cause of,For pulmonary thromboembolism risk factors;plasma plasminogen activator inhibitor-1 and coagulation fibrinolytic factors such as tissue factor abnormalities are common reasons.Conclusion The etiology Study of pulmonary embolism is great significance.for pulmonary embolism diagnosis treatment and prognosis.