Objective To analyze the acute cerebral watershed infarction by intravascular interventional therapy. Methods 36 patients with acute cerebral watershed infarction patients were given endovascular treatment. Results After treatment of patients with carotid artery stenosis score and neurological deifcits were signiifcantly improved after 24 months of follow-up, no recurrence or transient ischemic attack. Conclusion In strict grasp the operation indications on the basis for endovascular treatment of patients with acute cerebral watershed infarction, can effectively improve the patients with neurological impairment, the prognosis of recurrent disease.

Objective To analyze the application value of sodium ozagrel combined with aspirin in the treatment of cerebral infarction. Methods84 cases of patients with cerebral infarction were randomly divided into two groups, 40 cases in control group treated with sodium ozagrel in the treatment of the observation group, 44 cases in the control group based on the use of aspirin treatment.ResultsThe total efficiency of the observation group was significantly higher than the control group, neurological deficit score, ADL score after treatment were significantly better than the control group. Conclusion Sodium ozagrel combined with aspirin can promote the nerve function defect of infarction patients significantly improved, significant effect.

研究显示我国老年糖尿病的患病率为10%-20%,占总体糖尿病病人的60%以上。对于老年人来说,低血糖的危害非常大,可以诱发心律失常、心绞痛、心肌梗死、中枢神经系统的损害,如果不得以及时纠正可以危及生命。临床上低血糖症主要表现为反应性肾上腺素分

Objective To study the expression of aminoglutaminic acid transporter(EAAT) mRNA in cerebral cortex of diabetic rats having brain injury and its potential mechanism.Method Twenty-eight Wistar rats were randomly divided into two groups:14 rats fed with normal diet were selected as control group,and 14 rats fed with high glucose and high fatty diet plus intraperitoneal injecting STZ for inducing insulin resistance were selected as model group.Rats of two groups were free water-intake and illumination was given for 12h.One month late,blood glucose was measured.After 6 months,and EAAT-1,EAAT-2 and EAAT-3 mRNA expressions in cerebral cortex were detected by RT-PCR.Results The blood glucose level in model group[(20.016±1.984)mmol/L)]was higher than normal value(P<0.01) and that in control group[(3.82±0.123)mmol/L,P<0.01],The islet cells decreased significantly in pancreatic tissue in model group,while a lot of islet cells were observed in control group.The expression of EAAT-3 mRNA in model group was higher than that in control group(P<0.01).Conclusions The up-regulate expression of EAAT-3 could be an important factor in brain injury of rats with diabetes mellitus.

胰岛素抵抗是指胰岛素受体或受体后缺陷导致胰岛素在细胞中的信号障碍,使靶细胞对胰岛素的反应性下降或抵抗。这种胰岛素数量不少但质量差,或胰岛素抗体及升糖激素等物质产生过多,或者靶器官对胰岛素的反应性下降,引起胰岛素分泌不足,最终导致糖耐量异常或出现糖尿病。糖尿病人多同时伴发高血脂、高血压及肥胖等代谢综合征,其

Objective the purposes of this test is to study gene expression and mechanism concerning receptors of metabolic pattern(transporter of aminoglutaminic acid) in brain injured with diabetes.Methods There are twenty-eight Wistar rats to be randomly distributed class of A(this is control group breeding with routine animal feeds,the numbers are fourteen.) and B(this is test group breeding with high glucose and high fat animal feeds,in addition to inducing insulin resistance through injectioned STZ,the numbers are fourteen.).Drink and food of all rats is random,and receiving12 hours illumination cycle.A month later,all rats are be detected blood glucose.Six months later,the mRNA expression of EAAT-1、mGluR-4、mGluR-6 is be monitored with RT-PCR in pallium of diabetes' rats.Results the mRNA expression of mGluR-6 in class of B is more obviously increase than A(P0.05),however,the mRNA expressions of EAAT-1 and mGluR-4 is poor compared with class of A.Conclusion the increase of mGluR-6 may play a part in brain injured with diabetes.

本文对100例急性脑卒中病例进行回顾分析,发现急性脑卒中患者预后与外周血白细胞数量多少有关. 资料与方法 一般资料:收集我科2009年1月～2010年1月份患者100例,均符合全国第四届脑血管会议制订的脑卒中标准[1],并经头CT证实为新生的急性脑卒中患者.其中缺血性脑卒中72例,出血性脑卒中28例.其中白细胞升高组为50例,男32例,女22例,年龄:44～52岁.二组病例既往无糖尿病及严重肝肾病史.

青年人脑卒中(包括脑梗死)是指年龄在45岁以下的人发生的急性脑血管功能障碍,其发病率约占全部脑卒中病例的5%～10%.尽管发病率明显低于老年人,近年来,青年人脑梗死患者日益增多,给家庭及社会带来更沉重的负担.青年人脑梗死的病因与65岁以上老年人脑梗死的病因相比有很大不同.老年人多是在动脉硬化基础上发病,而青年人则由多种病因引起.

<正> 临床分析一般资料:100例患者皆为我科门诊或住院病人,年龄38～82岁;男46例,女54例。临床特点:表现为发作性眩晕,发作时视物旋转或倾倒,伴有恶心呕吐,不敢睁眼。26例有耳鸣及不同程度的听力下降;10例有视力障碍;20例有不同程度的头痛、头昏,其中3例头痛剧烈,颈部强

2008年1月-2008年7月本科对发病6 h内的30例急性脑梗死(ACI)患者给予尿激酶(UK)或发病3 h内患者给予重组组织型纤溶酶原激活物(r-tpA)静脉溶栓治疗,疗效满意,现报道如下.