This present study aims to explore the potential impact of cell phone radiation on innate immunity in mice. Ninety-six male BALB/C mice aged 2-3 weeks were randomly distributed into 4 groups as blank control, control, TD-SCDMA and LTE-Advanced respectively, with 32 mice in each group. Mice were designed to be exposed to cell phone radiation for 4-8 weeks. Eight mice in each group were taken out for measurement given exposure periods were 4, 6 and 8 weeks respectively. Cell biological technique was conducted to assess the chemotaxis of neutrophils, and a morphological method was performed for the detection of phagocytosis of neutrophil and macrophage, while microbiological means was carried out to test the relative activity of lysozyme in serum of mice. As a result, the chemotaxis ratio of neutrophils was with little statistical difference among the four groups given a shorter exposure period. However, the ratios in TD-SCDMA and LTE-Advanced groups were decreased significantly on the condition that the exposure period was more than 6 weeks. No statistical difference was observed among the four groups during the entire exposure period in terms of the chemotaxis index. Phagocytosis of the innate cells as neutrophil and macrophage showed little change in the two control groups during the whole experimental stages, while the percentage in the two treated groups decreased statistically, and this kind of reduction was prone to feature time dependence. The activities of lysozyme in TD-SCDMA and LTE-Advanced groups declined significantly, further to that, the impact was climbing paralleled with the prolonged duration. It could be deduced that radiation from cell phones could weaken innate immunity in experimental mice; moreover, this adverse effect was seemingly more severe as the radiation exposure continued.

The present research aims to explore the potential impact of mobile phone radiation on bone marrow haematopoietic stem cell in mice. 160 male BALB/C mice aged 3–4 weeks were randomly distributed into 4 groups as blank control, control, TD-SCDMA and LTE-Advanced respectively, with 40 mice in each group. Mice were designed to receive cell phone radiation for 2–8 weeks. 10 mice in each group were taken out for examinations given radiation exposure periods were 2, 4, 6 and 8 weeks respectively. Then terminal deoxynuclotidyl transferase-mediated dUTP Nick-end labelling technique was conducted to detect the apoptosis of stem cell, and morphological method was carried out for the examination of micronucleus and subpopulations of bone marrow haematopoietic stem cell, while enzyme immune technique was performed to analyse the subsets of T lymphocyte. As a result, no statistical differences were discovered on issues of apoptosis and micronucleus during the entire experimental period in the 2 sham radiation control groups. Notwithstanding similar results were seen in TD-SCDMA and LTE-Advanced groups after exposed to radiation for 2 or 4 weeks, apoptosis began to be enhanced and micronucleus to be increased given the mice experience longer exposure as 6 or 8 weeks. There were virtually negligible effects on subpopulations of stem cell, as well as on subsets of T lymphocyte of peripheral blood during the entire experimental period in the 4 groups. It could be concluded that long-term radiation from cell phone could induce apoptosis of haematopoietic stem cell in mice; moreover, it seems to stimulate the formation of micronucleus, yet with even negligible effect on the proportions of stem cell subpopulations and peripheral blood T lymphocyte subsets.

①目的调查女性阴道内正常菌群分布,筛选阴道微生态制剂种子菌.②方法 取健康体检女性阴道分泌物经镜检及五联卡检测后细菌培养,挑选优势菌落革兰染色后生化反应初步诊断,采用实时荧光定量PCR进行分子生物学鉴定;直方图描述阴道正常菌群频数分布,筛选种子菌.③结果 224份健康体检女性阴道分泌物,共分离并鉴定7种(392株)细菌,其中嗜酸乳杆菌123株,分离率54.9％(123/224);发酵乳杆菌98株,分离率43.8％(98/224);棒状杆菌59株,分离率26.3％(59/224);缓症链球菌37株,分离率16.5％(37/224);表皮葡萄球菌29株,分离率12.9％(29/224);马胃葡萄球菌28株,分离率12.5％(28/224);粪肠球菌19株,分离率8.5％(19/224).④结论 健康女性阴道菌群的优势菌为发酵乳杆菌、嗜酸乳杆菌及棒状杆菌,可作为阴道微生态制剂的候选种子菌株.

①目的调查细菌性阴道炎病原菌分布情况,为细菌性阴道炎疫苗研制提供实验依据.②方法 取门诊初步诊断为细菌性阴道炎患者标本常规镜检及五联卡检测,阳性标本细菌培养后进行生化反应及实时荧光定量PCR鉴定,直方图描述病原菌频数分布.③结果 168例细菌性阴道炎标本中共分离136株致病菌,主要包括凝固酶阴性葡萄球菌44株、大肠埃希菌38株,肺炎克雷伯菌26株.④结论 引起细菌性阴道炎的主要病原菌为凝固酶阴性葡萄球菌、大肠埃希菌和肺炎克雷伯菌.